The mystery is in the pump. the first thing you notice is the pump. then there is the buying. all that buying. it has to come from somewhere. It isn't me or you.
I am the best compelling evidence I know of. Read My lips. 1000 points of supplements gave me a certain vision. My river of deliverance.
No folic acid will not make a fol of you. nor will mr b12. nor will mr b3 combined with inositol. it is a complex issue. you have to eat beets. take zinc. all the bees. have to. otherwise you die like a dog. end of story. e. c. a. cla inositol hexaphosphate. yes. all of that. the jury is in. lost 60 livres in 6 months. gained health. it does work. I do not lie. well not about something this serious.
Read Julian Whitaker. Reversing Heart Disease. He ain't kidding. It works. Fan. diet. exercise. supplements. It ain't fiction. It is life saving. Fat kills. end of story.
If you smoke and take vitamin e, you will be 70% less susceptible to the big C. true fact. But don't smoke. works too.
In 1900 1 in 30 got cancer. 1 in 7 HD. today that is 1 in 5 get C and 1 in 4 die from HD. You can get back to 1900. Read and learn. I did it. So can you.
You will see lots of conflicting studies. The trouble with the conflicting info is the groups are often small and results misinterpreted. Often the results could be due to chance. One study on vit e said there was a 10% increase in death from taking E. What they said was that and increase from 4.5 deaths among 1000 people in 7 years to 4.95 deaths in the e group in same period was significant. Why could this be hooey? Because the margin of error among this type of group could be almost 0.35 deaths per 1000. or it could be more... And the test group had a core of sick miners from Virginia waiting to die from black lung!!!! not a good group to pick from. Also such studies suffer from what they call too drastic a marker ... death is a very simple but perhaps too simple a marker to use a criteria to base a decision as to the effectiveness of a drug or its dangers.. there is not gradation to judge dosage from easily..
And you cannot take just alpha tocopherol, and not dl-alpha either.... You have to take excess gamma to delta and excess delta to alpha, and tocotrienols. and you take d not dl. And you cannot take just E. it is not good. you have to take them all, or non at all. and some people have to take more than others because of biochemical individuality. And some people taking some drugs should not take too much e. so you have to watch studies. later that study got panned by a Harvard statistician, but it was too late. the damage was done.. the panic about e had set in.. you have to read 100 books and then think. ...and when you cure yourself of a serious disease by experimentation and talk to others who have done the same, then you can see that often the medical establishment could be deficient of vitamin and nutrition knowledge.. not all, but the sum total of their rx for lifestyle stuff is just not aggressive or pan knowledgeable or intensive enuff to make a significant difference to their patients.
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"One of the earliest and best-known cohort studies began in 1948 in Framingham, Massachusetts, and continued for more than twenty years. The Framingham study was designed to help identify the characteristics associated with coronary heart disease and other cardiovascular disorders. Over 5,000 people, all of whom were free of the ailment, were questioned extensively on their living habits and given a number of medical tests, including blood-pressure measurements, electrocardiograms, and a variety of blood analyses. The investigators found that certain characteristics—high blood pressure, cigarette smoking, and high serum cholesterol—were more prevalent among those who subsequently developed heart disease than among those who did not. Such characteristics are usually called risk factors, a term that denotes a statistical association but leaves open the question of causality. For example, the Framingham study tells us that people with high serum cholesterol are more likely to develop heart disease than those with low serum cholesterol, but it does not demonstrate conclusively that high serum cholesterol causes heart disease.
Through the use of complex statistical techniques, it is possible in epidemiological studies to determine whether certain characteristics that are associated with a disease are primary or secondary. The Framingham study, for example, found that obesity was associated with heart disease. However, obese people tend to have high blood pressure and high serum cholesterol, and, of course, they may also smoke. When the Framingham researchers took these characteristics into account, they found that obesity, as such, was a secondary characteristic that had only a weak association with the disease. In other words, people who are obese are not greatly at risk of heart disease unless they also have primary risk factors such as high blood pressure.
Cohort studies can provide high-quality information. The findings of the Framingham study and several other cohort studies of coronary heart disease are the basis for the current public-health programs aimed at reducing high blood pressure.
A major problem with this type of study is the large proportion of subjects that are lost to follow-up, particularly in the long-term studies. Cohort studies are expensive and time-consuming; and they are usually not suitable for studying rare diseases, because extremely large numbers of people would have to be included in order to provide a sufficient number of cases. Because of their expense, cohort studies are not suitable for testing highly speculative hypotheses.
There is a variant of the cohort study in which the information is not gathered prospectively. Instead of choosing a group of people and following their fortunes over a period of months or years, researchers sometimes attempt to reconstruct the history of a cohort using available records and personal interviews. Because they go back in time to look at people who may have been exposed to a suspected disease-causing agent, the term applied to this approach is retrospective cohort study. Such studies are often useful in assessing the risk to any group that has been exposed to a common health hazard; for example, workers who have been exposed to lead, patients who have been exposed to a particular type of medical radiation, or people living in the neighborhood of a chemical dump site. "
You significantly reduce your risk factors for CVD if you reduce your homocysteine, apolipoprotein a, triglycerides, oxidized cholesterol, to a certain extent total cholesterol, weight, sat fat intake, trans fat and hydrogenated oil intake, sugar and white bread intake, alcohol and cigarettes, and increase exercise, whole grain, fiber, vegetables and anti-oxidants, fruit, and leafy greens. Like by perhaps 80% depending on degrees of the above.
I believe you could reduce risk factors by 90% by exercise, weight and diet control, if certain judicious use is made of supplements and perhaps where indicated membrane stabilizing medication. This is in print in box car letters in more than one medical and naturopathic tome, and there are literally 10's of thousands of clinical success stories in MD/ND clinics across NA. Cooper, Whitaker, Pritikin, Erasmus, Rath, and many, many more.
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