It would appear that COQ10, vitamin E, and Vitamin C have a vital, mutually supportive role to play in reducing Lipid peroxidation and therefore in arterial plaque formation. How on earth then, a statin drug like Lipitor, which interferes with CoQ10 production as much as it interferes with the production of cholesterol in the liver, achieves a reduction in plaque formation, is beyond us.
Perhaps Statins such as Lipitor do not reduce lipid peroxidation, but just do not increase the rate of cholesterol plaquification much, allowing hard fibrous plaque to form but reducing the formation of soft, easily ruptured plaque. This would decrease the HA rate, but not interfere with the progress of artherosclerosis in total amount, so much as in quality.
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