Correct! Congratulations! Woohoo!! Finally! Now Here is Our Prize!
But, if you keep flattening the curve beyond what is essential, then you will kill more people with curve flattening than the virus will.
And this is the absurdity of what humans are attempting to do.
It is time to have the "talk" with everyone here. Because many have been following along, but have not been able to decrypt the complexities,,, and why both of us are persistently talking past one another.
Communicable diseases, in the community have been studied for ages:
There is no way you will stop the bulk of the world from pissing in the pool at one end, and not having the virus reach you at the other. Just ask Ebola. If persistent in a population with appropriate level of transmission and no means to kill virus, There really isn't jack shit the World can do. Ebola,,, hopped on a fucking plane and flew to NY. Measles? Yeah.. look up the last measles pandemic. We have an ebola vaccine. Have you gotten one? Dengue fever?
WHO estimates some .... 67% of the population is infected with HSV-1 yep. you know someone with Herpes. Lets all stop pretending about sex for a moment. Wanna hazard HPV, CMV? Has safe sex and lots of antiviral drugs cured the world of HIV? Still waiting. Same for HSV. Gancyclovir, Acyclovir Valcyclovir your ass until fucking Godot arrives. It is still here,,, getting a free ride, until every human on the planet, including Eric Trump,,, and maybe a few lurkers here,,, finally,,, have sex. (that was a joke,,, Eric Trump has children,,, we can surmise it was possible... ).
We are way past the containment phase.
There will be a significant reservoir of virus in the community for as long as we survive without a vaccination, or a drug comes along which kills the fuck out of the virus at a rate faster than it can transmit. End.of.fucking.story. Done. Put.a.fork.in.it.
HCQ isn't that drug, and whether or not your immune system is,,, vis a vis. "herd" immunity, or whatever that means,,,, is question of how much piss there is in the pool at any given time. Not pissing in the pool is pretty unlikely. There is always piss in the pool. 7.8 Billion fucking morons on this planet,,, there are bound to be a few fuckups.
Time to get back to work and learn how to not overbreathe, spit, or shit on your coworkers. It's worse than influenza it's serious shit and you may die.
We need to work on testing methods and ways in which we can adapt to detecting people who are asymptomatically carrying this disease, do everything we can to reduce transmission so that we do not spread it, and find a vaccine. Damn quick.
More sobering news:
Below is first the easy reader version followed by a reference paper. This information has been brewing for a while, This is the first peer reviewed version.
scmp.com
Coronavirus could attack immune system like HIV by targeting protective cells, warn scientists
Researchers in China and the US find that the virus that causes Covid-19 can destroy the T cells that are supposed to protect the body from harmful invadersOne doctor said concern is growing in medical circles that effect could be similar to HIV
nature.com
CorrespondenceOpen AccessPublished: 07 April 2020SARS-CoV-2 infects T lymphocytes through its spike protein-mediated membrane fusion Xinling Wang, Wei Xu, Gaowei Hu, Shuai Xia, Zhiping Sun, Zezhong Liu, Youhua Xie, Rong Zhang, Shibo Jiang & Lu Lu Cellular & Molecular Immunology (2020) Cite this article
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COVID-19, the novel coronavirus disease caused by SARS-CoV-2 and outbroken at the end of 2019 in Wuhan, China, 1 becomes a worldwide pandemic. SARS-CoV-2 belongs to the betacoronavirus genus and has 79.5% identity to SARS-CoV. SARS-CoV-2 uses angiotensin-converting enzyme 2 (ACE2) as its host entry receptor. 2 The clinical manifestations of COVID-19 include pneumonia, diarrhea, dyspnea, and multiple organ failure. Interestingly, lymphocytopenia, as a diagnostic indicator, is common in COVID-19 patients. Xiong et al. found upregulation of apoptosis, autophagy, and p53 pathways in PBMC of COVID-19 patients. 3 Some studies reported that lymphocytopenia might be related to mortality, especially in patients with low levels of CD3+, CD4+, and CD8+ T lymphocytes. 4, 5 Lymphocytopenia was also found in the Middle East respiratory syndrome (MERS) cases. MERS-CoV can directly infect human primary T lymphocytes and induce T-cell apoptosis through extrinsic and intrinsic apoptosis pathways, but it cannot replicate in T lymphocytes. 6 However, it is unclear whether SARS-CoV-2 can also infect T cells, resulting in lymphocytopenia. |
