SI
SI
discoversearch

We've detected that you're using an ad content blocking browser plug-in or feature. Ads provide a critical source of revenue to the continued operation of Silicon Investor.  We ask that you disable ad blocking while on Silicon Investor in the best interests of our community.  If you are not using an ad blocker but are still receiving this message, make sure your browser's tracking protection is set to the 'standard' level.
Biotech / Medical : GUMM - Eliminate the Common Cold

 Public ReplyPrvt ReplyMark as Last ReadFilePrevious 10Next 10PreviousNext  
To: out_of_the_loop who wrote (619)5/19/1999 11:59:00 AM
From: out_of_the_loop  Read Replies (2) of 5582
 
Zicam mechanism update

I reviewed my posts and they are factual but the Zicam mechanism of action as I described it is not complete, so in the interests of anal retentiveness, clarity and accuracy, here is more.

Zicam binds to the cells and gets into the cells. Zicam binds to the virus as well, just like Tremacamra, so it shares some features and has others that are different than "T"'s. Since Zicam binds to the cells, and gets in AND binds the virus, it can theoretically work on 3 levels:

1. It binds the virus before it can enter the cell (like T).
2. It occupies the binding site on the cell and prevents the virus from getting in.
3. It enters the cell and binds virus inside the cell, thereby decreasing its effectiveness in replicating (virus can't get into the protein-making machinery needed) and in getting out of the cell (because bound viral particles can't get out).

So, while I apologize for not being entirely complete, I think this clarification illustrates better why Zicam is likely to be more effective than "T" or anything else out there.
Report TOU ViolationShare This Post
 Public ReplyPrvt ReplyMark as Last ReadFilePrevious 10Next 10PreviousNext