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To: Neocon who wrote (294)	6/17/2000 10:45:00 PM
From: Father Terrence	Read Replies (1) \| Respond to of 1576

... and they are hesitating in contacting the CDC and NIH?????? This is outrageous.

To: Neocon who wrote (294)	6/17/2000 10:49:00 PM
From: Ilaine	Read Replies (1) \| Respond to of 1576

Are you saying that she has internal bleeding? Something like disseminated intravascular coagulation? The blood clots inappropriately, and then the clotting factor is exhausted, and she bleeds?

To: Neocon who wrote (294)	6/17/2000 11:04:00 PM
From: Ilaine	Read Replies (3) \| Respond to of 1576

The reason I am asking is that I am working on a case where a 12 year old boy died of side effects from Epstein-Barr virus, which doesn't ordinarily kill people. However, there are some rare, but well-known conditions which may result, a couple of which cause internal bleeding due to a pathological condition that prevents clotting. The internal bleeding is fatal. That child, too, had his kidneys shut down. So I can't help but wonder if the cases are somehow similar. I haven't yet determined whether there was malpractice, but the doctors were definitely at a loss. One treatment I have read about recently that they did not try looks promising: tPA, plasminogen activator. The doctors are now giving it for strokes, it helps prevent permanent damage if given soon enough. This doctor thinks it should be given for shock: Am Surg 2000 Mar;66(3):284-90 Related Articles, Books, LinkOut Traumatic shock alias posttrauma critical illness. Hardaway RM Department of Surgery, Texas Tech University Health Sciences Center, El Paso 79905, USA. Trauma is the most common cause of death under the age of 45. Many trauma patients die of multiple organ failure, especially acute respiratory distress syndrome. The basic cause of traumatic shock has only partially been elucidated. Data resources include research papers on the subject of trauma and shock from 1875 to the present. These papers numbered more than 40,000. Almost all of the papers proposed that traumatic shock was due to hypovolemia. The concept of a shock toxin as promulgated during World War I is correct. This toxin is a thrombogenic aminophospholipid that occurs only on the inner layer of all cell membranes and is liberated by cell destruction. It causes disseminated intravascular coagulation, which may obstruct the microcirculation of any and all organs producing multiple organ failure by microclots. These microclots may be lysed by plasminogen activator and circulation to the organs restored. ncbi.nlm.nih.gov