Silicon Investor (SI) -- The First Internet Community

STOCKTALK

We've detected that you're using an ad content blocking browser plug-in or feature. Ads provide a critical source of revenue to the continued operation of Silicon Investor. We ask that you disable ad blocking while on Silicon Investor in the best interests of our community. If you are not using an ad blocker but are still receiving this message, make sure your browser's tracking protection is set to the 'standard' level.

Strategies & Market Trends : Booms, Busts, and Recoveries -- Ignore unavailable to you. Want to Upgrade?

To: Moominoid who wrote (31953)	4/20/2003 2:49:15 PM
From: smolejv@gmx.net	Read Replies (1) \| Respond to of 74559

it's a typical case of a host-parasite coexistence. quote from the net: P. falciparum creates protein knobs on the surfaces of the red blood cells it attacks. These knobs attach the cell to the lining of the blood vessel, preventing its removal to the spleen for destruction. The parasite slows detection by the immune system by changing the makeup of the knobs periodically, substituting or rearranging its 150 “var” (variability) genes, a strategy unique to malaria. A pattern of remission and relapse results as the immune system learns each new “code” only to have it again changed. Patients with malaria gradually do develop immunity that modifies the course of the disease, but this immunity has a degree of strain specificity. iow, hangs out the sign "nobody home" and coasts along. Interesting the sickle cell anaemia makes it easier for humans to handle the side effects - possible evolutionary pressure from the parasite.