AIDS epidemics a tragic result of vaccine development?
The Origin of AIDS and HIV May Not Be What You Have Learned
originofaids.com
Most people believe that the origin of HIV, the AIDS virus, derives from some natural evolutionary event. Key among these HIV origin theories is the so called "cut hunter theory" in which a human, allegedly African native, received a bloody wound or infected splash while preparing a chimpanzee carrying a similar virus (i.e., SIVcpz). Most recent research, along with the scientific consensus, holds that the origin of HIV and AIDS could never have happened this way.
In 2001, The Royal Society of London's conference proceedings, which sought to determine the initial cause of AIDS and the origin of HIV, were published for the world to behold. The most highly respected scientists and academicians debated the possibility that HIV-1, the most widespread and deadly human AIDS virus, evolved from accidental vaccine contaminations and subsequent transmissions to mostly to African villagers....
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...According to scientific records,2 African chimpanzees were used in the manufacture of the HB (hepatitis B) vaccines during the early 1970s. Additional documents prove that human HB viruses cultured in vivo in chimpanzees were returned to humans whose infected blood serum was then pooled to develop four different strains of experimental HB vaccine pilot tested between 1970 and 1975 in New York City and central Africa. This HB vaccine theory of HIV zoonosis proposes that endogenous, or more likely exogenous, progenitor viruses were activated24 when serially transmitted from humans to chimpanzees, then back to humans. Subsequently, pooled blood serum containing HB surface antigen and/or live virions, a milieu ripe for viral recombination, was used to develop the four suspected vaccines administered to New York’s gay population and simultaneously to sub-Saharan Africans. Besides the phylogenetic evidence cited above, epidemiological evidence also supports this HB vaccine theory of HIV/AIDS origination.
Figure 1 is derived from Higginson and Muir’s report on cancer studies conducted by the International Agency for Research in Cancer (IARC) in collaboration with the National Cancer Institute (NCI).25 Figure 2 derives from this data superimposed on a map of HIV-1 seroprevalence in Africa reported by the U.S. Department of Commerce in a publication discussing desirable depopulation associated with HIV/AIDS.26 Additional evidence here was supplied in the chronology of the early hepatitis B vaccine trials compiled by Goodfield. 27 The two maps, juxaposed, show a striking correlation between hepatitis B vaccine and liver cancer experiments conducted in Africa during the early 1970s, and the countries in central and southern Africa with the high est HIV-1 seroprevalence rates by 1994. The black squares indicate areas participating in the HB cancer virus research and vaccine trials.
It should also be noted that Mozambique has one of the highest rates of HIV-2, which was allegedly discovered by Essex et al.,28 in Senegalese female prostitutes years after the African hepatitis B vaccination pilot studies began. Due to their state-authorized employment and high risk for infection, Senegalese female prostitutes were required to receive hepatitis B vaccinations for relicensure. That Essex et al. found SIVagm, a documented vaccine contaminant, in the blood of these human subject, is additionally compelling evidence in support of the HB vaccine AIDS origination theory.29
In brief, a well documented, theoretically viable, and generally neglected evolutionary route of SIVagm to HIV-1 zoonosis sequentially involves: 1) Polio vaccine recipients worldwide, including gay men in New York, and Blacks in Central Africa, were exposed to simian viruses including SV40, SFR (Simian Foamy Retroviruses containing reverse transcriptase), SIVagm, and perhaps others from the mid-1950s, through at least the 1960s;2,4 2) Between 1965 and 1970, researchers in NYC “isolated” and then inoculated the MS-2 strain of HB virus into the above cited New York and African HB vaccine study “volunteers.”2,30 3) Human derived HB viruses, and potentially activated retroviral sequences, were then transferred to chimpanzees, then back again to humans in NYC and central Africa during the development and testing of four genetically altered subtypes of the pre-1975 experimental HB vaccine.32,33 HIV-1 progenitor contamination, recombination, and/or transmission risks were likely increased during this process by: a) human incubation for more than a decade of polio vaccine contaminants and recombinants including SV40, SFR, and possibly SIVagm; b) the pooling of infected blood serum donated by hundreds of gay American and Black African polio vaccine recipients who had subsequently received injections with chimpanzee cultured strains of HB virus; c) the biohazardous laboratory conditions and viral containment problems reported by the HB vaccine investigators and their affiliates; and finally 5) The four pooled serum-derived HB vaccines that were administered to thousands of test subjects by 1975, primarily gay males in NYC and central African Blacks. This series of events provides the best explanation for an early to mid-1970s “punctuated origin event” most precisely fitting the etiological determinations of the HIV-1/AIDS pandemic.10
Again, it should be noted that the African “volunteers” inhabited a geographic area consistent with the highest rates of HIV-1 seroprevalence. Among the nations where rates are highest, HB studies were conducted in: Senegal, Cote d’Ivoire, Uganda, Kenya, Swaziland, and the northeastern part of South Africa. According to circumstantial evidence, eastern Zaire bordering the West Nile region of northwest Uganda also hosted such trials.2,25-27
Historic Precedence for the HB Vaccine Hypothesis
There is historic precedence for this precise HB thesis. According to Beale, the risk of HB viruses contaminating human blood serum and subsequent vaccinations was determined as early as 1942. Then, more than 62 deaths and 28,500 cases resulted from serum HB contaminated yellow fever vaccines.31
According to Hilleman, early yellow fever vaccines also delivered leukemic retroviruses to human populations due to caged animal and laboratory contaminations and concomitant vaccine transmissions.13
Dr. Hilleman additionally reinforced this “punctuated origin” thesis by describing the risks he encountered by importing contaminated African sub-human primates for vaccine research and development at the Merck pharmaceutical company. Between the late 1950s through the 1970s, Dr. Hilleman told Harvard medical historian Edward Shorter in 1987, “I brought African greens in. I didn’t know we were importing AIDS virus at the time.”13
Given these statements of fact, it is reasonable to suggest, as stated above, the earliest HB vaccine pilot studies may have activated an endogenous or exogenous HIV-related retroviral gene in one or more of the primates,24 fulfilling the “starburst phylogeny” antecedents advanced by Myers et al.10
During the Royal Society’s symposium on the origin of AIDS, Hooper’s 1950s OPV/AIDS hypothesis was largely rebuked because he failed to establish the use of chimpanzees by the Wistar Institute in the production of the suspected OPV.18 Moreover, this vaccine was not given selectively to New York’s gay male population. Curiously, Merck’s early 1970s hepatitis B vaccine trials that did involve gay men in NYC, and Blacks in central Africa, partially prepared in Litton Bionetics (LB) exported/Merck imported African chimpanzees, ironically went without mention....
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