Silicon Investor (SI) -- The First Internet Community

STOCKTALK

We've detected that you're using an ad content blocking browser plug-in or feature. Ads provide a critical source of revenue to the continued operation of Silicon Investor. We ask that you disable ad blocking while on Silicon Investor in the best interests of our community. If you are not using an ad blocker but are still receiving this message, make sure your browser's tracking protection is set to the 'standard' level.

Politics : Ask Michael Burke -- Ignore unavailable to you. Want to Upgrade?

To: Pogeu Mahone who wrote (112918)	4/17/2008 10:44:45 AM
From: Knighty Tin	Respond to of 132070

Serious article and study, but I can't get over the Ph.d's name on the study. Byung-Ho. Sounds like the star of a gay porno film. <G>

To: Pogeu Mahone who wrote (112918)	4/17/2008 1:11:38 PM
From: Skeeter Bug	Read Replies (3) \| Respond to of 132070

zeus, CRP is an effect, not a cause. the valid question is what causes the silent inflammation that results in increased levels of CRP. saying folks with elevated CRP have a higher risk of heart disease is like saying someone a light bulb that gives off heat has a higher chance of being on. well, duh! the elevated CRP is a direct result of elevated inflammation which is the ultimate cause of heart disease. the smart folks aren't making silly and obvious correlations, rather, they have moved up stream to try and identify the ultimate root cause of the inflammation that both elevates CRP and is a large cause of heart disease. heart disease doesn't appear to have much to do with dietary cholesterol as aspirin, which is an anti-inflammatory and has zero impact on cholesterol, is the medication that lowers the incidence of heart disease. it is an inflammatory condition. the drug companies make billions controlling cholesterol... so they forced that interpretation on the medical community. the key to avoiding elevated CRP levels in the first place is to eliminate silent inflammation by balancing anti-inflammatory eicosanoids and pro-inflammatory eicosanoids. also, tg/hdl (which indicates elevated levels of insulin) is a better predictor of silent inflammation than CRP as CRP only becomes elevated after acute illness is already present. frankly, i'd like to avoid the illness and the elevated CRP that goes with it instead of wait for elevated CRP and then try and manage my illness. iow, i macromanage wellness so i minimize the risk i have to micromanage illness. waiting for CRP to elevate is like waiting for a mortgage crisis to address the lending problems. it is too late then. catch it early and avoid elevated CRP altogether. for a good discussion on the topic: books.google.com the summary is that elevated insulin, almost always caused by a high glycemic load diet, coupled with a diet rich in excess omega 6 fatty acids. causes long term silent inflammation. this explains why those who eat lots of fruits and veggies (low glycemic load diet) have the lowest rates of cancer and why no micronutrients in the fruits and veggies have been found to have significant cancer lowering effects. it isn't the micronutrients as so many assumed, rather, it was the absence of a high glycemic load, and the associated elevated insulin levels, that reduces the incidence of cancer. remember, low glycemic load = lower silent inflammation. this should be 100% intuitive. humans were either designed or evolved to eat fruits, veggies, nuts and meat. massive industrialized high glycemic load complex carb factories didn't exist until very, very, very recently.