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Politics : President Barack Obama -- Ignore unavailable to you. Want to Upgrade?

To: Mac Con Ulaidh who wrote (65731)	11/27/2009 4:37:18 PM
From: Wharf Rat	Read Replies (2) \| Respond to of 149317

Maybe the pentagon studies related to IED induced brain damage. (That's good) Maybe some black-ops nerve gas study( that's bad). There can be reasons related to the military. Could we win a war by dosing the enemy? (That's very good) Would it be better to dose cats or soldiers or rats first? Da Rat will go with Da Mouse, but not if they want to stick needles in my brain. Effects of LSD on Cortex of Cat Correlated with Changes in Vital Signs THOMAS W. LANGFITT, M.D.; LOUIS A. FINNEY, M.D. AMA Arch Neurol. 1959;1(3):269-281. Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings. Introduction In a previous report15 it was demonstrated that large doses of lysergic acid diethylamide (LSD) had no consistent effect on primary sensory evoked potentials in the cortex of the cat. However, in a recent publication Purpura21 reported qualitative differences in the effect of LSD on cortical evoked potentials and postulated that these were due to anatomical differences in the termination of afferents in the cortex, some ending on soma and others on dendrites. Sensory afferents end primarily on the soma of cortical neurons. In contrast, direct electrical stimulation of the cortex produces a predominantly surface-negative wave, which is generally considered to represent the activity of apical dendrites.5,8,10 Furthermore, it is also thought to be a postsynaptic potential analogous to the postsynaptic potential of motoneurons in the spinal cord.9 Also, anatomical evidence has been advanced to indicate that callosal fibers terminate primarily on dendrites or as . . . [Full Text PDF of this Article] Author Affiliations Baltimore From the Division of Neurological Surgery, The Johns Hopkins University, School of Medicine. Present address (Dr. Finney): U. S. Naval Hospital, Chelsea, Mass. Presented in part before the American Physiological Society, London, Ont., Canada, September, 1958. Footnotes Accepted for publication March 2, 1959. Work performed under Department of Army contract. Aided by a grant from the National Institute of Neurological Diseases and Blindness, National Institutes of Health, U. S. Public Health Service. archneur.ama-assn.org