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Strategies & Market Trends : 2026 TeoTwawKi ... 2032 Darkest Interregnum -- Ignore unavailable to you. Want to Upgrade?

To: Pogeu Mahone who wrote (183484)	2/2/2022 8:20:29 PM
From: TobagoJack	1 Recommendation Recommended By Pogeu Mahone Read Replies (1) \| Respond to of 217986

in the meantime, long-covid might lead to Parkinson's even as folks are so rushing into Omicron around most of planet, having been earlier 'vaxx-ed' with possibly troublesome liquids the circle is tightening bbc.com How past pandemics may have caused Parkinson's In the 1960s, epidemiologists studying the long-term prognosis of survivors of the 1918 Spanish Influenza began to notice an unusual trend. Those who were born between 1888 and 1924 – meaning they were either infants or in young adulthood at the time of the pandemic – appeared to have been two or three times more likely to develop Parkinson’s disease at some point in their life than those born at different times. It was a striking finding. For while the potential neurological consequences of flu infections have been documented by doctors for centuries – there are medical reports of this which date back to 1385 – the sheer scale of the Spanish Flu, which infected around 500 million people globally, meant scientists could link a heightened risk of disease to the pandemic. In recent years, an elevated risk of Parkinson's has also been identified in the survivors of outbreaks of HIV, West Nile virus, Japanese encephalitis, Coxsackie, Western Equine virus and the Epstein-Barr virus. Neurologists attempting to understand why this happens believe that each of these viruses are capable of crossing into the brain, and in some cases damaging the fragile structures which control the co-ordination of movement, known as the basal ganglia, initiating a process of degeneration which can lead to Parkinson's. Now scientists are keen to monitor whether the current pandemic will also trigger a higher rate of Parkinson's cases in decades to come. "We don't know but we need to consider that this could become the case," says Patrik Brundin, a Parkinson's researcher at the Van Andel Institute, in Grand Rapids, Michigan. "There are several studies highlighting that people who have recovered from Covid often have long-term central nervous system deficits including loss of sense of smell and taste, brain fog, depression, and anxiety. The numbers are troubling." While Sars-CoV-2 can invade brain tissue, the scientific jury remains open on whether it will contribute to neurodegenerative disease. Coronaviruses are generally known as "hit and run viruses", because they tend to cause fairly short disease, even if this proves deadly in some cases. In contrast, DNA viruses such as Epstein-Barr can linger permanently in the body and are more associated with long-term illness. But there have been some indications in the past there might be more to coronaviruses than we perhaps suspect. In the 1990s, Canadian neurologist Stanley Fahn published a study which identified antibodies to the coronaviruses that cause the common cold in the cerebrospinal fluid of Parkinson's patients. Over the past year, scientists like Brundin have been concerned by the emergence of a small handful of case studies describing patients who have developed what doctors term acute Parkinsonism – abnormalities such as tremors, muscle stiffness, and impaired speech – following Covid-19 infection. More research has found that some Covid-19 patients have disruptions in one of the body's most critical systems, known as the kynurenine pathway. This runs from the brain to the gut, and is used to produce a number of crucial amino acids required for brain health. But when it malfunctions, it can lead to the accumulation of toxins which are thought to play a role in Parkinson's disease. Other neurologists, however, warn it is still far too early to draw any link between Covid-19 and Parkinson's.